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METHYL-SP for Liver and Gallbladder Detoxification Support
- Compounds for healthy homocysteine metabolism
- Supports healthy phase II methylation during
liver detoxification reactions
- Supports bile synthesis and metabolism
Methylation reactions are
crucial steps in the phase II, detoxification pathways. This product provides the major nutrients and cofactors
necessary to support methylation reactions. It also provides methyl donors that help to lower homocysteine levels
and improve bile synthesis.
Dosage: - 2-3 capsules 3x
day.
Ingredients:Vitamin C 50 mg
Vitamin E 20 IU
Vitamin B6 10 mg
Folic Acid 1200 mcg
Vitamin B12 600 mcg
Magnesium 140 mg
Choline (soy) 50 mg
Trimethylglycine 550 mg
MSM 50 mg
Beet root 50 mg
Betaine HCl 60 mg
Other Ingredients: Magnesium citrate, choline bitartrate, ascorbic acid, alpha tocopheryl acetate, vegetarian capsule
(HPMC), rice flour, pyridoxal-5 phosphate, methylcobalamin.
RESEARCH:
This product helps support healthy methylation
reactions. Methylation is the biochemical reaction responsible for the transfer of one-carbon molecules. This chemical
reaction is a crucial step in many important biochemical pathways and is dependent on essential nutrients to act
as cofactors. Altered methylation reactions have been associated with elevated homocystiene, dementia, depression,
compromised Phase II detoxification, birth defects, etc. This product should be considered by all women that use
oral contraceptives because it contains the essential nutrients that are depleted while on the birth-control pill.1 2 3 4 5 6 7 8
1 Brattstoerm L, et al. Plasma homocysteine
in women on oral oestrogen-containing contraceptives and in men with oestrogen-treated prostatic carcinoma. Scand
J Clin Lab Invest. 52, 1982:283-287.
2 Smithells RW, et al. Possible prevention of neural-tube defects by periconceptional vitamin supplementation.
Lancet, Feb 16, 1980:339-342.
Butterworth CE, et al. Folate deficiency and cervical dysplasia. JAMA. 1992;267:528-533.
3 Lindenbaum J, et al. Neuropsychiatric disorders caused by cobalamin deficiency in the absence of anemia or macrocytosis.
N Eng J Med. 1988;318:1720-1728.
4 Allen RH, et al. Serum beatine, N,N-dimethlyglycine and N-methylglycine levels inpatients with cobalamin deficiency
and related inborn errors of metabolism. Metab. 1993;42(11):1448-1460.
5 Zingh JM, Jones PA. Genetic and epigenetic aspects of DNA menthylation on genome expression, evolution, mutation
and carcinogenesis. Carcinogenesis. 1997;18(5):869-882.
6 Smythies JR. The role of the one-carbon cycle in neuropsychatric disease. Biol Psychiatry. 1984;19(5):755-758.
7 Finkelstein JD. Homocysteine: a history in progress. Nutr Rev. 2000;58(7):193-204.
8 Krumdieck CL, Prince CW. Mecahnisms of homocysteine toxicity on connective tissues: implications of morbidity
of aging. J Nutr. 2000;130:365S-368S.
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